Center-Authored Papers
Filters: Author is Rabinovitch, Peter S [Clear All Filters]
Array-based comparative genomic hybridization in ulcerative colitis neoplasia: single non-dysplastic biopsies distinguish progressors from non-progressors.. Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc. 23(12):1624-33. Abstract
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2010.
Ulcerative colitis-associated colorectal cancer arises in a field of short telomeres, senescence, and inflammation.. Cancer research. 71(5):1669-79. Abstract
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2011.
Dietary supplement use and risk of neoplastic progression in esophageal adenocarcinoma: a prospective study.. Nutrition and cancer. 60(1):39-48. Abstract
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2008.
Dietary supplement use and risk of neoplastic progression in esophageal adenocarcinoma: a prospective study.. Nutrition and cancer. 60(1):39-48. Abstract
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2008.
Ulcerative colitis is a disease of accelerated colon aging: evidence from telomere attrition and DNA damage.. Gastroenterology. 135(2):410-8. Abstract
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2008.
Ulcerative colitis-associated colorectal cancer arises in a field of short telomeres, senescence, and inflammation.. Cancer research. 71(5):1669-79. Abstract
.
2011.
Array-based comparative genomic hybridization in ulcerative colitis neoplasia: single non-dysplastic biopsies distinguish progressors from non-progressors.. Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc. 23(12):1624-33. Abstract
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2010.
mTOR is a key modulator of ageing and age-related disease.. Nature. 493(7432):338-45. Abstract
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2013.
mTOR inhibition alleviates mitochondrial disease in a mouse model of Leigh syndrome.. Science (New York, N.Y.). 342(6165):1524-8. Abstract
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2013.
Ulcerative colitis is a disease of accelerated colon aging: evidence from telomere attrition and DNA damage.. Gastroenterology. 135(2):410-8. Abstract
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2008.
Ulcerative colitis-associated colorectal cancer arises in a field of short telomeres, senescence, and inflammation.. Cancer research. 71(5):1669-79. Abstract
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2011.
No telomere shortening in marrow stroma from patients with MDS.. Annals of hematology. 88(7):623-8. Abstract
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2009.
No telomere shortening in marrow stroma from patients with MDS.. Annals of hematology. 88(7):623-8. Abstract
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2009.
Ulcerative colitis is a disease of accelerated colon aging: evidence from telomere attrition and DNA damage.. Gastroenterology. 135(2):410-8. Abstract
.
2008.
Ulcerative colitis is a disease of accelerated colon aging: evidence from telomere attrition and DNA damage.. Gastroenterology. 135(2):410-8. Abstract
.
2008.
Dietary supplement use and risk of neoplastic progression in esophageal adenocarcinoma: a prospective study.. Nutrition and cancer. 60(1):39-48. Abstract
.
2008.
mTOR inhibition alleviates mitochondrial disease in a mouse model of Leigh syndrome.. Science (New York, N.Y.). 342(6165):1524-8. Abstract
.
2013.
mTOR is a key modulator of ageing and age-related disease.. Nature. 493(7432):338-45. Abstract
.
2013.
Genetic variation in prostaglandin E2 synthesis and signaling, prostaglandin dehydrogenase, and the risk of colorectal adenoma.. Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology. 19(2):547-57. Abstract
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2010.
Clonal expansions in ulcerative colitis identify patients with neoplasia.. Proceedings of the National Academy of Sciences of the United States of America. 106(49):20871-6. Abstract
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2009.
Reduction of age-associated pathology in old mice by overexpression of catalase in mitochondria.. The journals of gerontology. Series A, Biological sciences and medical sciences. 63(8):813-22. Abstract
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2008.
Ulcerative colitis is a disease of accelerated colon aging: evidence from telomere attrition and DNA damage.. Gastroenterology. 135(2):410-8. Abstract
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2008.
Polyploidy, aneuploidy and the evolution of cancer.. Advances in experimental medicine and biology. 676:1-13. Abstract
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2010.
Deletion at fragile sites is a common and early event in Barrett's esophagus.. Molecular cancer research : MCR. 8(8):1084-94. Abstract
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2010.
Dietary supplement use and risk of neoplastic progression in esophageal adenocarcinoma: a prospective study.. Nutrition and cancer. 60(1):39-48. Abstract
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