Center-Authored Papers
Filters: Author is Denny, Joshua C [Clear All Filters]
Use of diverse electronic medical record systems to identify genetic risk for type 2 diabetes within a genome-wide association study.. Journal of the American Medical Informatics Association : JAMIA. 19(22):212-218. Abstract
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2012.
Genome- and phenome-wide analyses of cardiac conduction identifies markers of arrhythmia risk.. Circulation. 127(13):1377-85. Abstract
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2013.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
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2014.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Use of diverse electronic medical record systems to identify genetic risk for type 2 diabetes within a genome-wide association study.. Journal of the American Medical Informatics Association : JAMIA. 19(22):212-218. Abstract
.
2012.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Return of individual research results from genome-wide association studies: experience of the Electronic Medical Records and Genomics (eMERGE) Network.. Genetics in medicine : official journal of the American College of Medical Genetics. Abstract
.
2012.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Use of diverse electronic medical record systems to identify genetic risk for type 2 diabetes within a genome-wide association study.. Journal of the American Medical Informatics Association : JAMIA. 19(22):212-218. Abstract
.
2012.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Genome- and phenome-wide analyses of cardiac conduction identifies markers of arrhythmia risk.. Circulation. 127(13):1377-85. Abstract
.
2013.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
Inactivating mutations in NPC1L1 and protection from coronary heart disease.. The New England journal of medicine. 371(22):2072-82. Abstract
.
2014.
High density GWAS for LDL cholesterol in African Americans using electronic medical records reveals a strong protective variant in APOE.. Clinical and translational science. 5(5):394-9. Abstract
.
2012. Doing Business with Arnold Library
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